GYNAECOMASTIA
Overview
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Benign proliferation of glandular tissue in the male breast
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Pseudogynaecomastia - Excessive development of the male breast from subareolar fat deposition without glandular proliferation
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Bilateral in up to 75%
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Aetiology​
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Idiopathic​
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Physiologic
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Neonatal​
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Pubertal
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Elderly
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Pathologic​
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Increased oestrogen or reduced testosterone
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Chronic renal or liver disease
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HIV
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Pharmacologic
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Antiandrogens​
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Antibiotics - Isoniazid, ketoconazole, metronidazole
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Antiulcer - Cimetidine, ranitidine, proton pump inhibitors
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Chemotherapy - Alkylating agents, methotrexate, vinca alkaloids
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Cardiovascular - Digoxin, verapamil, diltiazem, nifedipine, amiodarone, captopril, enalapril
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Psychoactive - Diazepam, antipsychotics, antidepressants
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Drugs of abuse - Marijuana, alcohol, amphetamines, heroin, methadone
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Histologic classification​
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Florid​ - Increased ducts and cellular stroma​​
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Intermediate
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Fibrous - Extensive stromal fibrosis, minimal duct proliferation
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Patient assessment
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History
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Age of onset​
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Duration
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Symptoms
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Pain​
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Lumps
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Galactorrhoea
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Past medical history (see above)​
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Drug history
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Anticoagulants​
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Risk factors for wound healing
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Cause of gynaecomastia
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Social history
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Recreational drug use (see above)
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Smoking history
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Classification
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Simon classification
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Grade I - Small enlargement without skin excess​
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Grade IIa - Moderate enlargement without skin excess
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Grade IIb - Moderate enlargement with skin excess
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Grade III - Marked enlargement with excess skin, mimicking female breast ptosis
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Management
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Treatment or removal of cause
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Surgical treatment:​​
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Liposuction
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Excision of excess glandular tissue and repositioning of nipple areola complex:
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Principles in line with breast reduction
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Incisions
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Periareolar incision​
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Transaxillary incision
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Free nipple grafting
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Allows en bloc resection of skin and breast tissue​
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Risk of graft failure and consequent nipple loss
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